6-Nitroindoline is a reagent used in chemical synthesis. It has been used in the synthesis of 5-HT1 receptor agonists, 5-HT2C/2B receptor antagonists, serine protease inhibitors and vanilloid receptor ligands.

7-(Cyanomethoxy)indole is a reagent for chemical synthesis. It’s a reagent in the creation of N-benzyl dihydroindole LTD4 antagonists.

7-Azaindole is a reagent in the synthesis of pharmaceuticals.

7-Benzyloxyindole is a reagent for the preparation of inhibitors like HCV inhibitor and pyrazolo quinolone derivatives that function as adenosine receptor antagonists.

7-Bromoindole is a reagent for chemical synthesis.

7-Fluorooxindole is a reagent for chemical synthesis. It has been used in the synthesis of anxiety and bipolar treating compounds.

Azelaic acid is a dicarboxylic acid found in cereals and animal products. It is bactericidal against Proprionibacterium acnes and Staphylococcus epidermidis by inhibiting synthesis in microbial cellular proteins. It is found to be useful in controlling several skin conditions, including acne and hyperpigmentation. In melanoma cells azelaic acid inhibits DNA synthesis, leading to a significant reduction in proliferation.

beta-D-Glucose pentaacetate is a sugar that can be used as a reagent in chemical synthesis. In pancreatic islets, the secretory response to L-leucine is increased by beta-D-Glucose pentaacetate, increasing insulin secretion in both diabetic and normal rats.

Cysteamine Hydrochloride or cysteamine is a mercaptoethylamine hydrochloride salt reductant that forms in mammals from pantetheine formation by degrading Coenzyme A. It has been used as a cystine depleting agent as well as a somatostatin inhibitor.

Cysteamine has been shown in vivo to remove accumulated cystine in lysosomes by forming disulfide bridges with cystine. The interaction between cysteamine and cystine forms cysteine and cysteine-cysteamine mixed disulfide which can freely exit lysosomes.

Cysteamine has also been shown to inhibit somatostatin however, the mechanism of action has not been elucidated. It is believed to either inhibit somatostatin precursor formation or by promoting somatostatin degradation.